Alleged Chlorine Attack Checklist
When another attack with chlorine gas is reported in Syria, it might help to check the evidence. This page is intended as a resource for that. It's a very basic start for now.
The color should be pale green-yellow, or may appear yellow, or even white if thin enough. It should not be neon green, orange, or reddish-brown.
Clinical signs, or more commonly symptoms, are the bio-medical indicators that help decide what poison might be responsible. Most of them are non-specific - can be caused by many different things. None can prove or disprove a certain poison, but enough consistent signs with no contrary signs is a strong indication of that poison.
The following table can be copied and filled in with reported or seen symptoms, and any alternate guess of comparison. Columns can be added for different classes of victims, etc. As used on other pages here:
|Chlorine Clinical Signs Comparison Table|
|Known indicator||Reported indicator||Shown indicator||alternate cause?|
|dyspnea (rapid, difficult breathing)||
|other skin burns|
|Weakness in the legs|
- Mechanism: Chlorine reacts with water in and out of the body to form hydrochloric acid and hypochlorous acid. Both are extremely poisonous. (Medlineplus)
- A good catalog of expected effects was described to Doctors without Borders (MSF) and Amnesty International following the March 16, 2015 alleged chlorine attack in Sarmin.
- CDC: During or immediately after exposure to dangerous concentrations of chlorine, the following signs and symptoms may develop: ◦Blurred vision
- Burning pain, redness, and blisters on the skin if exposed to gas. Skin injuries similar to frostbite can occur if it is exposed to liquid chlorine
- Burning sensation in the nose, throat, and eyes
- Chest tightness
- Difficulty breathing or shortness of breath. Thesemay appear immediately if high concentrations of chlorine gas are inhaled, or they may be delayed if low concentrations of chlorine gas are inhaled.
- Fluid in the lungs (pulmonary edema) that may be delayed for a few hours
- Nausea and vomiting
- Watery eyes
- Medlineplus (partial)
- Breathing difficulty (from breathing in the chlorine)
- Throat swelling (may also cause breathing difficulty)
- Water filling the lungs (pulmonary edema)
- Loss of vision
- Severe pain in the throat
- Severe pain or burning in the nose, eyes, ears, lips, or tongue
- Blood in the stool
- Burns of the food pipe (esophagus)
- Severe abdominal pain
- Vomiting blood
- Holes (necrosis) in the skin or tissues underneath
- The French troops in the path of the gas cloud sustained about 6,000 casualties. Many died within ten minutes (primarily from asphyxia and tissue damage in the lungs), and many more were blinded. Chlorine gas forms hypochlorous acid when combined with water, destroying moist tissue such as the lungs and eyes. The chlorine gas, denser than air, quickly filled the trenches and forced the troops out into heavy enemy fire.
- The first effect of inhalation of chlorine is a burning pain in the throat and eyes, accompanied by a sensation of suffocation; pain, which may be severe, is felt in the chest, especially behind the sternum. Respiration becomes painful, rapid, and difficult ; coughing occurs, and the irritation of the eyes results in profuse lachrymation. Retching is common and may be followed by vomiting, which gives temporary relief. The lips and mouth are parched and the tongue is covered with a thick dry fur. Severe headache rapidly follows with a feeling of great weakness in the legs; if the patient gives way to this and lies down, he is likely to inhale still more chlorine, as the heavy gas is most concentrated near the ground. In severe poisoning unconsciousness follows; nothing more is known about the cases which prove fatal on the field within the first few hours of the "gassing," except that the face assumes a pale greenish yellow colour.
- When a man lives long enough to be admitted into a clearing station, he is conscious, but restless; his face is violet red, and his ears and finger nails blue ; his expression strained and anxious as he gasps for breath; he tries to get relief by sitting up with his head thrown back, or he lies in an exhausted condition, sometimes on his side with his head over the edge of the stretcher in order to help the escape of fluid from the lungs. His skin is cold and his temperature subnormal; the pulse is full and rarely over 100. Respiration is jerky, shallow and rapid, the rate being often over 40 and sometimes even 80 a minute ; all the auxiliary muscles come into play, the chest being over-distended at the height of inspiration and, as in asthma, only slightly less distended in extreme expiration. Frequent and painful coughing occurs and some frothy sputum is brought up. The lungs are less resonant than normal, but not actually dull, and fine riles with occasional rhonchi and harsh but not bronchial breathing are heard, especially over the back and sides. Headache is generally severe, and there is also considerable epigastric discomfort, due partly to the strain of coughing and partly to gastric irritation, as it is increased if an attempt is made to eat.
- The intense dyspncea of this asphyxial stage lasts about thirty-six hours, after which it gradually subsides, if death does not occur before. The patient, exhausted from his fight for breath, then falls asleep and wakes up feeling much relieved. A few hours later acute bronchitis or broncho-pneumonia develops. In severe cases the quiescent interval is short and the bronchitis very severe. The sputum is now viscid, yellow or greenish, and muco-purulent with occasional streaks of blood. Respiration becomes more shallow and rapid, and the rate may finally be even 70 or 80 a minute. The pulse is small and very rapid ; the temperature rises, and is often as high as 104. The patient may now become delirious. Pleurisy may occur, and in some instances empyema and gangrene of the lung have followed.
- After recovery from the bronchitis and pneumonia the patient remains weak and exhausted for a considerable time. He gets tired very rapidly and is unable to walk quickly or up hill without getting short of breath, even after the last signs of bronchitis have disappeared. He may continue to have attacks of dyspncea and cyanosis for several weeks. The frightful experience he has passed through often affects his nervous system, and some of the attacks are doubtless aggravated by apprehension. Headache, vertigo and dyspepsia may continue for several weeks.
- The First World War > Strategy and Tactics > Chlorine Gas – Spartacus Educational
- Chlorine gas destroyed the respiratory organs of its victims and this led to a slow death by asphyxiation. One nurse described the death of one soldier who had been in the trenches during a chlorine gas attack. “He was sitting on the bed, fighting for breath, his lips plum coloured. He was a magnificent young Canadian past all hope in the asphyxia of chlorine. I shall never forget the look in his eyes as he turned to me and gasped: I can’t die! Is it possible that nothing can be done for me?” It was a horrible death, but as hard as they tried, doctors were unable to find a way of successfully treating chlorine gas poisoning.
- Chlorine is a greenish-yellow, noncombustible gas at room temperature and atmospheric pressure. Its intermediate water solubility accounts for the effect on the upper airway and lower respiratory tract. Prolonged exposure to chlorine gas may occur because its moderate water solubility delays onset of upper airway symptoms for several minutes. In addition, the density of the gas is greater than that of air, causing it to remain near ground level and increasing exposure time. The odor threshold for chlorine is approximately 0.3-0.5 parts per million (ppm); however, distinguishing toxic air levels from permissible air levels may be difficult until irritative symptoms are present. As the concentration of chlorine gas exposure increases, the severity of symptoms and rapidity of onset increase. Concentrations above 400 ppm are often fatal.
See also Tekkim Chemical Test Video for what might be the effects of chlorine on caged rabbits.
- Grass and vegetation exposed to chlorine also become exposed to acid due to its water content. Green plants directly exposed will be bleached pale yellow afterwards.
- In some cases, a magenta or re-purple colored liquid and staining is seen in conjunction with an event that probably involved genuine chlorine gas. This can be synthesized by mixing the magenta powder Potassium Permanganate and liquid hydrochloric acid. This is alleged to be done inside barrel bombs dropped by government forces. However, that's questionable. This is the method used in the likely chlorine gassing of rabbits in 2012 (see above)